查詢結果分析
來源資料
相關文獻
- Vasoconstrictor Produced from Jejunum during Postprandial Hyperemia
- 家蠶最後一齡幼蟲之前胸腺腺苷酸環化酶之活性變化
- Determination of Adenosine and Cordycepin Contents in Cultivated Cordyceps by Micellar Electrokinetic Capillary Chromatography
- 基因治療的困境:1996年的回顧評論
- 血液中腺苷酸專一性高壓液相層析定量研究
- 蛋白激酶C調控腺苷酸環化酶第六亞型之機制及生理作用
- 養殖臺灣蜆於不同貯藏溫度下腺苷三磷酸及其相關化合物與游離胺基酸之變化
- Alleviation of Hemorrhagic Shock-Induced Hepatic Adenosine Triphosphate Depletion by Enterectomy in Rats
- Effects of Adenyl Purines in Human Uterine Arteries and Uterine Myometrium
- Content of Adenylate Nucleotides and Energy Charge in the Early Stage of Germination of Orthodox and Recalcitrant Seeds
頁籤選單縮合
題 名 | Vasoconstrictor Produced from Jejunum during Postprandial Hyperemia=食後充血時空腸產生的血管收縮因子 |
---|---|
作 者 | 甯學寒; 巴正豐; 肖雲; 周欽城; | 書刊名 | 慈濟醫學 |
卷 期 | 9:1 1997.03[民86.03] |
頁 次 | 頁19-26 |
分類號 | 415.121 |
關鍵詞 | 血管收縮因子; 動脈張力; 食後充血; 5-羥色胺受體; 腺苷; Vasoconstrictor; Arterial ring tension; Postprandial jejunal hyperemia; 5-HT receptor; NECA; |
語 文 | 英文(English) |
中文摘要 | 為驗證,進食後的小腸充血可能產生某種因子而導致血管回饋效應這一假設,在11隻犬上,收集了於腸腔含生理鹽水、消化食物、或腺苷模擬劑5'-(N-ethylcarboxamide)adenosine(NECA)時的游離空腸段靜脈血。用克氏液灌流的離體犬腸系膜上動脈(N=7)或大鼠主動脈(N=6)環,來觀察等張張力變化以測試血漿的活性。在狗腸腔內放置鹽水、食物、和NECA時,空腸血流分別為61±9.4,124±38,及159±31mL/min/100g腸重(平均值±標準差)。灌食物和NECA時的血漿使動脈環張力分別增加0.73±0.14g(N=14)和1.42±0.23g(N=15),遠高於灌鹽水時的血漿所致(0.17±0.04g. N=24)。直接注入NECA灌流液,血管環張力只下降或無變化。以phenoxybenzamine和SQ-29548分別阻斷α-腎上腺能受體和前列腺素受體,均未影響灌食物及灌NECA時血漿的活性。食後充血時的血漿尚能增加大鼠主動脈環的張力(分別為0.18±0.03g和0.56±0.05g),其遠高於灌鹽水時的血漿所致(0.09±0.02g, N=6/組)。以cyproheptadine阻斷5-羥色胺受體,大鼠動脈環的張力為阻斷前的-4.2±16.7%和11.9±15.3%(p<0.01)。以上結果提示,食物或腺苷引起空腸充血時,有一種血管收縮因子釋放入空腸靜脈血漿,該因子可能與5-羥色胺樣物質有關。 |
英文摘要 | To determine the hypothesis that postprandial intestinal hyperemia may induce some factors to cause feedback response in the vessels, samples of venous plasma was taken from canine jejunal segments when the lumen contained normal saline (Pl□), digested food (Pl□), or 5'-(N-ethylcarboxamide) adenosine (Pl□), an agonist of adenosine, in 11 dogs. The vasoactivity of the plasmas was tested by the measurement of isometric tension of isolated rings of canine superior mesenteric artery (N=7) or rat aorta (N=6) perfused with Krebs-Ringer bicarbonate. The canine jujunal blood flow was 61±9.4, 124±38, and 159±31 mL/min/100 g gut wt (mean±SE), respectively, during luminal placement of normal saline, food, and NECA. Pl□ and Pl□ increased the arterial rings’ tension more than pl□ did (0.73±0.14g, N=14; 1.42±023g, N=15; and 0.17±0.04g, N=24 respectively). Direct injection of NECA into the perfusate had no effect on the rings’ tension. Phenoxybenzamine (an α–adrenergic receptor blocker) and SQ-29548 (a thromboxane A₂ receptor blocker) did not affect the change in ring tension produced by Pl□ or Pl□ Pl□ and Pl□ also increased rat aortic rings’ tension more than Pl□ did (0.18±0.03g, 0.56±0.05g, and 0.09±0.02g respectively, N=6, each group). Cyproheptadine (a serotonin receptor blocker), decreased the rat ring tensions induced by Pl□ and Pl□ to -4.2±16.7% and 11.9±15.3% of the pre-blockade values, respectively (p<0.01). The results suggest that a vasoconstrictor is released into jejunal venous plasma during the food-and NECA-induced jejunal hyperemia in dogs. The vasoconstrictor may be a serotonin-like substance. |
本系統中英文摘要資訊取自各篇刊載內容。