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題 名 | 種用菜鴨飼料中缺乏吡哆醇或葉酸對於菜鴨胚胎發育之影響 |
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作 者 | 胡怡浩; 陳保基; | 書刊名 | 畜產研究 |
卷 期 | 28:2 1995.06[民84.06] |
頁 次 | 頁91-100 |
分類號 | 437.75 |
關鍵詞 | 褐色菜鴨; 缺乏吡哆醇; 缺乏葉酸; 胚胎異常; Brown tsaiya duck; Pyridoxine deficiency; Folic acid deficiency; Abnormal embryo; |
語 文 | 中文(Chinese) |
中文摘要 | 本研究旨在探討半純化種用菜鴨飼料中缺乏?哆醇或葉酸,對於菜鴨胚胎發育之影響。試驗期計廿九週分二階段進行,前十五週菜鴨飼養於地面,後十四週菜鴨飼養於網狀鴨床上。試驗分四組,前三組分別為缺乏?哆醇、缺乏葉酸及對照組,飼予半純化玉米澱粉-酪蛋白飼料,第四組飼予正常之玉米-大豆粕飼料。 產蛋褐色菜鶼被飼予僅需要量30%之?哆醇或葉腸飼眼,在試驗第二階段?哆醇缺乏使母鴨產蛋率下降至0;葉酸缺乏則並未發現對產蛋之影響。二項維生素之缺乏對受精率之的影響在二階段中未有一致之結果。在試驗第一階段鴨隻孵化率在缺乏?哆醇組、缺乏葉酸組、對照組及正常組,分別為68.3%、67.3%、85.8%及78.7%,缺乏組極顯著較對照及正常組低10.4~18.5%;試驗第二階段各組孵化率分別為65.7%、57.2%、68.6%及67.2%,缺代組較對照及正常組雖仍低1.5~11.4%,然僅缺乏葉酸組達顯著性,結果顯示缺乏葉酸組之胚胎死亡率顯著較高。四組縱異常率在第一階段分別為0.60%、0.93%、0.6%及0.67%,四組間差異不顯著;試驗第二階段異常率分別為2.0%、7.2%、1.0%及1.2%,試驗第二習段結果,雖顯非缺乏組異常率較高,然僅缺乏葉酸組達顯著性。缺乏?哆醇或葉酸鴨胚胎死亡率在孵化期第四週及未出殼發生率較高。試驗中缺乏?哆醇之異常率不高,鴨胚胎症狀以曲趾及上喙短或缺為主;缺乏葉酸之鴨胚胎異常症狀依發生頻度多寡,依序為曲趾、上喙短或缺、頭部出血、滑腱症、喙嫌窄及皮下出血。試驗結果顯示,缺乏?哆醇影響產蛋率及孵化率;缺乏葉酸對孵化率及鴨胚胎有不良之影響。 |
英文摘要 | This study was conducted to understand the effects of dietary pyridoxine or folic acid deficiency on embryonic development of Tsaiya breeder. The ducks were raised on floor during the first fifteen weeks of this experiment. They were raised on wire during the last fourteen weeks of the experiment. Ducks were divided into four groups. The first three groups were pyridoxine deficient, folic acid deficient, and control group, all were fed on corn starch-casein diet. The fourth group was fed a normal corn-soybean diet. The Brown Tsaiya ducks were supplied 30% of the requirements of pyridoxine or folic acid fed the deficient diet. For pyridoxine deficiency, the egg production dropped to zero in the second stage, but there was no effect of folic acid deficiency on egg production. The results of the influence on fertility was not consistent in the two stages. For pyridoxine deficient, folic acid deficient, control, and normal group, the hatchabilities were 68.3%, 67.3%, 85.5% and 78.7%, respectively, in the first stages. Which hatchabilities of deficeint groups were significantly lower than the control and normal groups. Which were 65.7%, 57.2%, 68.6% and 67.2%, respectively, in the second stage. Although the hatchabilities of both deficient groups were lower than those of the control and normal groups, but significantly only between the folic acid deficient and control or normal groups. The result showed that the mortalities in the folic acid deficient group were significantly higher than those in the control and normal groups. In the experiment, the peak of embryonic mortality occurred in the fourth week with many ducklings unhatched in the pyridoxine or folic acid deficient groups. The embryonic abnormalities were 0.6%, 0.93%, 0.60%, and 0.67%, respectively, in the first stage. They were not different significantly; they were 2.0%, 7.2%, 1.0%, and 1.2%, respectively, in the second stage but significantly only between folic acid deficient and control or normal groups. The result showed that the abnormality of the second stage were higher than that of the first stage, especially in the deficient group. The embryonic abnormality of the pyridoxine deficient and control or normal groups. The results showed that the abnormality of the second stage were higher than that of the first stage, especially in the deficient groups. The embryonic abnormality of the pyridoxine deficient group was not as high as that of folic acid deficient group. The major symptoms of embryonic abnormalities were curled webs and shorter or absent upper beak. For folic acid deficiency, the major abnormal symptoms, according to the descendent frequency of occurrence, they were curled webs, shorter or absent upper beak, haemorrhage on head, perosis, narrowed beak, and subcutaneous haemorrhage. The results of this experiment showed the egg production and hatchability were engatively influenced in pridoxine dificient groups, and they were severely engatively influenced on hatchability and embryonic development in the folic acid deficient group. |
本系統中英文摘要資訊取自各篇刊載內容。