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題 名 | No Association between TAP1 DpnⅡ Polymorphism and Bronchopulmonary Dysplasia=TAP1基因DpnⅡ多形性和早產兒肺支氣管發育不全無關 |
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作 者 | 林鴻志; 蘇百弘; 許欽木; 周宜卿; 巫康熙; 林曉娟; 蔡輔仁; 蔡長海; | 書刊名 | 臺灣兒科醫學會雜誌 |
卷 期 | 46:6 民94.11-12 |
頁 次 | 頁341-345+403 |
分類號 | 417.5171 |
關鍵詞 | 抗體表現相關運輸體; 基因多形性; 聚合酶鏈反應; 個案對照研究; TAP; Gene polymorphism; Bronchopulmonary dysplasia; Polymerase chain reaction; Case-control study; |
語 文 | 英文(English) |
英文摘要 | The possibility that a family history of asthma may have a role in susceptibility to bronchopulmonary dysplasia (BPD) had been raised in several reports, and there was evidence of a strong association between transporter associated with antigen processing (TAP1) polymorphism and asthma in Taiwanese population. To test whether TAP polymorphism has a role in the BPD, we investigated the association between TAP1 polymorphism and BPD by analyzing the results of genotype distribution. The study included 224 ventilated preterm infants (<30 weeks) who had respiratory distress syndrome (RDS) and needed intermittent mandatory ventilation (IMV) during Jan. 1999 to July 2003. The typing of TAP1 polymorphism was performed by polymerase chain reaction (PCR)-based restriction analysis. The demography between two groups of these ventilated preterm infants was not different. We observed no significant differences in genotype distribution or allele frequency of the TAP1 polymorphisms between BPD and their respective control infants. There was also no significant difference in genotype distribution of the TAP1 polymorphism with duration of IMV. Therefor, we conclude that TAP1 polymorphism is not a useful marker for predicting the susceptibility or severity to BPD for Taiwanese. |
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