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頁籤選單縮合
題 名 | 非胰島素依賴型糖尿病的致病機轉=Pathogenesis of Non-insulin-dependent Diabetes Mellitus |
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作 者 | 陳國東; 陳建仁; 傅茂祖; 王躬仁; | 書刊名 | 中華民國家庭醫學雜誌 |
卷 期 | 8:4 1998.12[民87.12] |
頁 次 | 頁169-180 |
分類號 | 415.668 |
關鍵詞 | 非胰島素依賴型糖尿病; Non-insulin-dependent diabetes mellitus; Pathogenesis; Insulin-resistance; |
語 文 | 中文(Chinese) |
中文摘要 | 世界上有5%的人罹患非胰島素依賴型糖尿病(non-insulin-dependent diabetes mellitus, NIDDM),它是人類重要的健康問題。β-細胞,橫紋肌,或肝臟對葡萄糖的代謝無法發揮功能,就有可能造成葡萄糖耐性不良或糖尿病的發生。目前的知識認為胰島素分泌不足或對胰島素產生拮抗性是NIDDM的主要致病機轉。不管是何種機轉引發糖尿病的生成,最後這兩種現象都會在NIDDM病人身上發現。它們兩者互為因果,究竟何者為原始原因,尚有待進一步研究。基因研究是未來探討NIDDM病因的重要方向。 |
英文摘要 | Non-insulin-dependent diabetes mellitus (NIDDM) is a major health problem, affecting 5% of the world population. Defects at the level of the β-cell, muscle, or liver can lead to the development of glucose intolerance or even overt diabetes mellitus. The concept has been evidenced that there are two primary defects responsible for the pathogenesis of NIDDM. In some NIDDM patients the primary defect starts at the level of the β-cell and manifests itself as an impairment in insulin secretion. In other NIDDM patients, the primary defect start as an impairment in tissue sensitivity to insulin. In either case diminished insulin secretion or insulin resistance, it initiates the development of NIDDM and will subsequently lead to the emergence of the second abnormality. In fact, both defects must be present simultaneously for moderate to severe glucose intolerance to occur. Genetic studies are an important direction to understand the pathogenesis for NIDDM in the future. |
本系統中英文摘要資訊取自各篇刊載內容。