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題 名 | A SPECT Study of Patients with Gait Apraxia Without Evidence of Frontal Lobe Dysfunction=單光子電腦斷層掃描研究顯示步態失用症的額葉功能正常 |
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作 者 | 陳仁澤; 傅中玲; 陳昭境; 劉仁賢; 單定一; 廖光淦; | 書刊名 | 中華醫學雜誌 |
卷 期 | 61:4 1998.04[民87.04] |
頁 次 | 頁216-222 |
分類號 | 415.81 |
關鍵詞 | 基底核; 額葉; 步態失用症; 單光子電腦斷層掃描; Basal ganglia; Frontal lobe; Gait apraxia; Single photon emission computed tomography; |
語 文 | 英文(English) |
中文摘要 | 背景 步態失用症的致病機轉迄今仍然不明。 方法 我們使用單光于電腦斷層掃描,來研究這類患者的大腦功能,以間接性的方法,了解 該疾病是否源於局部性大腦功能失調。 結果 共十六名患者(15男、1女;平均年齡70.5歲)參與研究。臨床表徵:9名(56.25 ﹪平衡失常,16名(100﹪),凍結步態,4名(25﹪)寬步伐,10名(62.5﹪)短步距,4 名(25﹪)失智,3名(18.75﹪)錐體徑路症狀,2名(12.5﹪)額葉症狀,6名(37.5﹪) 失禁。腦部磁振造影結果:4名(25﹪)白質軟化,3名(18.75﹪)腦室變大、3名 (18.75﹪)額葉病灶,1名(6.25﹪)頂葉病灶,16名(100﹪)隙空洞,其中基底核16 名,視丘6名。單光子電腦斷層掃描顯示:血流減少現象於額葉有3名,枕葉有 1名、基 底核有9名,視丘7名。雖然不正常單光子電腦斷層掃描有9名,但腦皮質的局部血流的P 值0.0613,基底核的局部血流P值0.0576,仍未具統計意義。 結論 本研究顯示步態失用症的額葉功能仍屬正常,其致病機轉仍待進一步研究。 |
英文摘要 | Background. The pathogenesis of gait apraxia (GA) is unknown. Even though imaging studies provide excellent assessment of brain morphology, there is still a lack of congruous results. Single photon emission computed tomography (SPECT) using Tc-99m hexamethylpropyleneamine oxime (HMPAO) may show alterations in regional cerebral blood flow (rCBF) and provide indirect information about brain metabolism. Methods. We conducted a SPECT study of GA patients and evaluated the related cortical function. rCBF was assessed in 16 GA patients (15 male, one female; age range 65-79 years, mean 70.5 years) by SPECT using HMPAO. Mean HMPAO cortical or basal ganglia/ cerebellum activity ratios were calculated. The regions of interest included the frontal lobe, parietal lobe and basal ganglia. A battery of CA tests and magnetic resonance imaging (MRI) of the brain were also performed in these 16 patients. Results. Nine of the patients had equilibrium disorder, and all 16 patients had locomotion disorder. The MRI findings were lacunar infarct (16/16 in basal ganglia, or 6/16 in thalamus), leukoaraiosis (4/16), enlarged ventricle (3/16), frontal lesion (3/16) and parietal lesion (1/16), Lower rCBF was noted in the frontal lobe (3/16), occipital lobe (1/16), thalamus (7/16) and basal ganglia (9/16). Though SPECT showed decreased rCBF in nine patients (9/16), mean cortical and basal ganglia regional uptake ratios in the patient group were not significantly different from values in the control group (cortical p=0.0613; basal ganglia p=0.0576, by Student's t-test). Conclusions. Though only a small number of patients were studied, it was clear that brain SPECT and MRI did not show any significant abnormalities in the frontal or parietal lobes of patients with GA. Thus, the pathogenesis of GA and its related anatomic lesion should be further investigated. |
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