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頁籤選單縮合
題 名 | 多重器官衰竭的致病機轉=Pathogenesis of Multiple Organs Failure |
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作 者 | 黃崇旂; 蔡熒煌; | 書刊名 | 中華民國重症醫學雜誌 |
卷 期 | 4:1 2002.01[民91.01] |
頁 次 | 頁42-49 |
分類號 | 415.2 |
關鍵詞 | 多重器官衰竭; 全身性發炎反應; 細菌內毒素轉移; Multiple organ system failure; Systemic inflammatory response syndrome; Bacterial-toxin translocation; |
語 文 | 中文(Chinese) |
中文摘要 | 隨著重症加護醫學的進步,多重器官衰竭已成為現在加護病房病人最大的死亡原因。其確定的定義現在尚未有一致性的共識,最常見的是發生在敗血症、外傷、燒傷、嚴重的發炎、急性呼吸窘迫症候群,或是各種的休克造成組織灌流不足。其致病機轉相當複雜,包括了體液性和細胞性的發炎介質,造成全身性的發炎反應。而且當某一器官遭受到傷害後,往往是多個病理機轉經由多條路徑,形成一個網狀相互作用。由於在疾病不同階段,體内防禦系統可能是被過度活化或是可能受到抑制,使得要針對這些免疫系統發炎反應的機轉來治療亦相當困難。 |
英文摘要 | With the advancement of intensive care medicine, multiple organ system failure (MOSF) has become the most common cause of death among ICU patients. Major risk factors for MOSF include sepsis, severe infection, trauma, burn, ARDS, and hypoperfusion from various types of shock. The pathogenesis of MOSF is very complicated, including humoral and cellular inflammatory mediators, leading to the systemic inflammatory response syndrome (SIRS). Once the organ injury has developed, multiple pathogenetic pathways occur simultaneously, forming an interactive network. Natural defense systems may become pathologically hyperactive, or suppressed at different stages in critically illness, making it difficult to target therapies directed at the immunoinflammatory mechanisms. |
本系統中英文摘要資訊取自各篇刊載內容。