查詢結果分析
相關文獻
- Urinary Endothelin-1 in Patients with Renal Disease
- Urinary Endothelin-1 in Children with Acute Renal Failure of Tubular Origin
- Increased Urinary Endothelin-1 Excretion in Newly Diagnosed Type 2 Diabetic Patients
- 內皮素和糖尿病腎臟病--基礎研究
- 內皮素(Endothelins)與糖尿病腎病變臨床研究
- 尿液內皮素
- Endothelin-1 Enhances Superoxide and Prostaglandin E2 Production of Isolated Diabetic Glomeruli
- cDNA Cloning and Expression Analysis of the Human UDPglucose Dehydrogenase
- 尿路結石之飲食治療與護理指導
- Orbital Rhinocerebral Mucormycosis Associated with Diabetic Ketoacidosis: Report of Survival of a 10-Year-Old Boy
頁籤選單縮合
題 名 | Urinary Endothelin-1 in Patients with Renal Disease=腎臟病人尿中之內皮素 |
---|---|
作 者 | 朱宗信; 吳明修; 謝博生; | 書刊名 | 臺灣醫學會雜誌 |
卷 期 | 97:10 1998.10[民87.10] |
頁 次 | 頁667-672 |
分類號 | 415.74 |
關鍵詞 | 腎臟病人; 尿; 內皮素; Endothelin-1; Glomerulonephritis; Chronic renal failure; β[feaf]-microglobulin; Renal tubule; Sodium; |
語 文 | 英文(English) |
英文摘要 | The purpose of this study was to investigate the usefulness of urinary endothelin-1 (ET-1) as a marker of renal disease. We measured urinary excretion of ET-1 in 28 patients with glomerulonephritis (GN), 22 patients with chronic renal failure (CRF), 40 patients with end-stage renal disease (ESRD) and 17 healthy volunteers. There was no significant difference in 24-hour urinary ET-1 excretion among the four groups (mean±SEM, 0.49±0.22 ng in controls, 0.79±0.37 ng in GN patients, 0.39±0.18 ng in CRF patients, and 0.28±0.11 ng in ESDR patients). The 24-hour urinary excretion of ET-1 in patients with GN or CRF showed significant correlation with the urinary excretion of sodium (r=0.27, p<0.05). The 24-hour urinary β��-microglobulin (β�烘) excretion in patients with CRF (18.4±2.6mg) or ESRD (9.7±1.1 mg) was significantly higher than in normal control subjects(0.23±0.11 mg). Serum creatinine concentration was positively correlated with the 24-hour urinary excretion of β�烘in patients with GN or CRF (r=0.50, p<0.001). These findings indicate that urinary ET-1 is not as good a marker of renal disease as urinary β�烘. However, it may be responsible for urinary sodium excretion in patients with GN or CRF. |
本系統中英文摘要資訊取自各篇刊載內容。