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題 名 | 抗磷脂抗體症病患併發慢性硬腦膜下出血:病例報告=Antiphospholipid Antibody Syndrome with Chronic Subdural Hemorrhage: A Case Report |
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作 者 | 周詠富; 顏威彰; 戴家煌; 官大紳; 洪章仁; | 書刊名 | 臺灣復健醫學雜誌 |
卷 期 | 35:2 2007.06[民96.06] |
頁 次 | 頁111-116 |
分類號 | 415.695 |
關鍵詞 | 抗磷脂抗體症; 硬腦膜下出血; 抗牛心脂素抗體; 抗凝固狼瘡; Antiphospholipid antibody syndrome; Subdural hemorrhage; Anticardiolipin antibody; Lupus anticoagulant; |
語 文 | 中文(Chinese) |
中文摘要 | 抗磷脂抗體症(antiphospholipid antibody syndrome)患者體內會有抗牛心脂素抗體 (anticardiolipin antibody) 和抗凝因狼瘡 (lupus anticoagulant) 這兩種自體免疫抗體存在,此類抗體會增加病患缺血性中風及反覆性腦部缺血的機率,而較少見出血的症狀發生,因此一般會長期給予病患口服抗凝血劑以預防缺血性中風發生。本病例為36歲女性,自民國87年確定診斷為抗磷脂抗體症後,即長期口服抗凝血劑來抑制栓塞產生,但是仍在這數年之內發生數次缺血性中風,經長期復健治療病情穩定後,病患轉送至護理之家繼續照顧,之後卻出現慢性硬腦膜下出血 (subdural hemorrhage) 的表現,再度住院治療,病患嚴重失能,其日常生活已完全需人協助,復健計畫著重於長期照顧及避免跌倒發生。本文將進一步探討抗磷脂抗體症造成病患反覆中風和其他併發症發生的病生理機轉,並針對本病例硬腦膜下出血的可能原因以及相關文獻記載做討論。 |
英文摘要 | Patients who suffer antiphosphlipid antibody syndrome may have two kinds of antibodies, Anticardiolipin and Lupus anticoagulant. These antibodies may increase the risk of ischemic stroke and cerebral ischemia. However, the incidence of cerebral bleeding is relative rare. As a result, patients with antiphosphlipid syndrome often take oral anticoagulants for a long-term period to prevent ischemic stroke attack. We report a female patient who was diagnosed antiphosphlipid antibody syndrome in 1998, and took oral anticoagulants for a long period to restrain the symptom of thrombosis. The INR(International Normalized Ratio)value has been kept between 1.5 to 2.0 by controlling the dosage of oral anticoagulants in the past years. However, she still suffered ischemic stroke several times during these years despite of using preventive oral anticoagulants. Left hemiparesis and motor aphasia occurred of repeated ischemic strokes, she could only communicate in a simple way and perform activity of daily activities with minimal assistance although she had received a long-term rehabilitation program. In the last ischemic stroke episode, she was admitted to the medical wards and received an adjustment of the dosage of oral anticoagulants to keep the INR range between 3.0 to 4.0 to efficiently avoid ischemic stroke. After being medically stable, she was transferred to our rehabilitation wards for further management of impaired cognitive function and mobility. After receiving rehabilitation programs, the patient had some impairment, and was still unable to be independent. Therefore she was transferred to a nursing home. Unfortunately, seizure attacked after discharge, and she was readmitted for treatment. Due to chronic subdural hemorrhage, a complication of oral anticoagulants, occurred. Antiplatelet agents replaced oral anticoagulants for the prevention of repeated ischemic stroke. No more new neurological symptoms happened after using antiplatelet agents. During the hospitalization, she received rehabilitation programs for improving her functional status and communication ability. But severe neurological deficits still cause her to lose almost all the abilities of daily living. The rehabilitation plans focused on long-term care and the prevention of falling. This article investigates the pathophysiology mechanism of repeated stroke and other complications caused by antiphospholipid antibody syndrome. On the other hand, we discuss the possible reasons of subdural hemorrhage in this case and some related bibliography. |
本系統中英文摘要資訊取自各篇刊載內容。