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題 名 | Modulatory Effect of Blood Cells on Hypoxic Vasoconstriction Response and Nitric Oxide Release in Rat Lungs=血球對缺氧之老鼠肺引起之血管收縮及一氧化氮釋出之調節作用 |
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作 者 | 高尚志; 魏崢; 黃志平; 劉美文; 江俊松; 汪大衛; 陳幸一; | 書刊名 | 臺灣醫學會雜誌 |
卷 期 | 98:1 1999.01[民88.01] |
頁 次 | 頁39-44 |
分類號 | 415.121 |
關鍵詞 | 血球; 缺氧; 肺; 血管收縮; 一氧化氮釋出; 調節作用; Lungs; Hypoxia; Blood cells; Nitric oxide; |
語 文 | 英文(English) |
英文摘要 | In this study, we investigated the modulatory effects of different types of blood cells on hypoxic pulmonary vasoconstrictive (HPV) response and nitric oxide (NO) release in isolated rat lungs. The lungs were perfused at a constant flow with physiologic saline solution (PSS). The changes in pulmonary arterial pressure (PAP) and NO release were observed. Two hypoxic challenges with a 5% CO□-95% N□ gas mixture were carried out in each experiment. Hypoxia induced pulmonary vaso- constriction, as reflected by an increase in PAP (0.88 ± 0.22 cmH□O). At the same time, NO (342.9 ± 78.3 mv) release from the lungs was also increased. Addition of white blood cells (WBCs, 0.70 to 0.88 * 10□/mL), platelets (1.48 to 1.96 * 10□ /mL), or red blood cells (RBCs, 4.6 to 6.6 * 10□/mL) into the perfusate produced different effects on PAP and NO changes. WBCs decreased the pulmonary vasoconstriction response and this was accompanied by an increase in NO release. Platelets had no significant effects on either PAP or NO. RBCs significantly potentiated the PAP increase and attenuated the NO release. The results indicate that NO release during hypoxia tends to offset pulmonary vasoconstriction and that NO release and HPV response are modulated by different cell elements. |
本系統中英文摘要資訊取自各篇刊載內容。