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頁籤選單縮合
題 名 | Hypokalemia and Paralysis=低血鉀性癱瘓 |
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作 者 | 林石化; 許燿東; 海裴林; | 書刊名 | Journal of Medical Sciences |
卷 期 | 22:1 2002.02[民91.02] |
頁 次 | 頁1-12 |
分類號 | 415.597 |
關鍵詞 | 低血鉀性; 癱瘓; Hypokalemia; Paralysis; |
語 文 | 英文(English) |
英文摘要 | Hypokalemic paralysis (HP) is a potentially reversible electrolyte and muscle disorder in emergency medicine. A vigorous search of the underlying etiology is mandatory to avoid missing a treatable cause. The measurement of blood and urine electrolytes and acid-based parameters can provide valuable clues to simplify different diagnosis. In patients with hypokalemic periodic paralysis (HPP) due to increased transcellular shift of potassium ions (K+) into cells, there is usually no acid-base disturbance, a low rate of K+ excretion and a very low transtubular potassium gradient (TTKG). Family history of paralysis, hypernatremia, clinical symptoms or signs related to hyperthyroidism, and drug histories are important keys to differentiate the subgroups of HPP. In patients with non-HPP, there is excessive excretion of K+ in association with an acid-based balance abnormality and a high TTKG. Hence non-HPP patients can readily be divided into two groups; one with hyperchloremic metabolic acidosis and the other with hypochloremic metabolic alkalosis. Assessment of urine ammonium excretion will subdivide the former group whereas blood pressure, plasma renin activity and aldosterone concentrations will clarify the causes of the latter group. In patients with HPP, the dose of KCl should be as small as possible to avoid rebound hyperkalemia. In contrast, patients in the non-HPP group with excessive renal K+ excretion need large doses of KCl to correct the severe deficit of a total body K+. |
本系統中英文摘要資訊取自各篇刊載內容。