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題名 | Carbon Dioxide Poisoning--Case Report and Literature Review=二氧化碳中毒--病歷報告及文獻回顧 |
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作者 | 沈修年; 余忠仁; | 書刊名 | 胸腔醫學 |
卷期 | 16:1 2001.03[民90.03] |
頁次 | 頁28-34 |
分類號 | 418.735 |
關鍵詞 | 二氧化碳中毒; CO[feaf]intoxication; Carbon dioxide intoxication; |
語文 | 英文(English) |
中文摘要 | 民國八十九年六月,四名工人在一棟大樓地下變電所做焊接工程時,因意外觸動二氧化碳自動滅火系統,高濃度的二氧化碳導致四名工人在逃跑中昏迷,並且造成最後救出的一名工人死亡;另外,在二氧化碳中毒(carbon dioxide intoxication)後存活的病人中,也觀察到短暫的實驗檢查及肺部影像異常的現象。在此,除了詳細的病歷報告之外,我們也回顧了二氧化碳對動物及人體的各種影響。二氧化碳的致昏效應(narcotic effect),在二百多年前即是已知的現象;高濃度二氧化碳對呼吸及心血管系統有抑制作用,濃度百分之八十的二氧化碳在三十秒內即可使實驗動物的呼吸終止,並造成等電位型態的腦波圖(isoelectric electroencephalography)。因此,與一般認知不同的是,吸入高濃度二氧化碳導致死亡的原因並非單純的缺乏氧氣,而是因為二氧化碳中毒所致。 |
英文摘要 | IN June 1999, an automatic fire-extinguishing system accident caused carbon dioxide (CO2) poisoning and resulted in the immediate collapse of four men. Three of the four men survived, the fourth did not. Details of the case histories of the two survivors are presented here. We also reviewed related literatures on the various effects of high-concentration CO2 on both animals and human beings. The narcotic properties of CO2 have been known for nearly 200 years. High concentration CO2 can have a depressant effect on both respiratory and cardiovascular systems. Inspired CO2 levels of up to 80% of gas mixture can cause respirator arrest and isoelectric electroencephalograph (EEG) patterns within 30 seconds. Contrary to popular belief, the cause of death in breathing high-concentration CO2 is not hypoxia, but carbon dioxide poisoning. Among CO2 poisoning survivors, transient abnormalities in the laboratory test data and pulmonary images can be observed. |
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