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相關文獻
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題 名 | 支氣管氣喘的致病機轉=Pathogenesis of Bronchial Asthma |
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作 者 | 顏鴻欽; | 書刊名 | 國防醫學 |
卷 期 | 31:4 2000.10[民89.10] |
頁 次 | 頁279-288 |
專 輯 | 支氣管性氣喘專輯 |
分類號 | 415.425 |
關鍵詞 | 哮喘; 致病機轉; 呼吸道發炎; Asthma; Pathogenesis; Airway inflammation; |
語 文 | 中文(Chinese) |
中文摘要 | 支氣管氣喘是一種反覆發作的呼吸道慢性發炎疾病。大部份的支氣管氣喘病人有明顯的遺傳傾向。同時,兒童型支氣管氣喘常合併異位性體質,異位性體質是目前已知的支氣管氣喘最明顯的潛在因子。有此體質的病人在碰到過敏原時,會產生IgE抗體媒介的免疫發炎反應而導致氣喘的發作。呼吸道慢性發炎的特性是診斷、治療、以及預防支氣管氣喘的主要根據。支氣管氣喘的免疫組織病理特徵包括被活化的肥胖細胞及其他呼吸道細胞釋放出多種不同的趨炎介質。這些發炎介質吸引許多其他發炎細胞的聚集,包括嗜中性白血球、嗜伊紅白血球以及TH2為主的淋巴球,而導致更厲害的發炎。呼吸道發炎會導致呼吸道反應過度、造成急性支氣管收縮、呼吸道水腫、黏液栓子形成、氣流阻塞以及呼吸困難的症狀。厲害的發炎反應會造成呼吸道上皮細胞的脫落、膠原蛋白在基底膜下的沈著等病理變化,同時不完全的呼吸道修復過程會導致持續性的呼吸道通氣障礙。 |
英文摘要 | Bronchial asthma is a recurrent chronic inflammatory disorder of the airways. Most of the patients with asthma have strong family history. Atopy, an IgE-mediated response to common aeroallergens, is the strongest predisposing factor for developing asthma and is frequently seen in child-onset asthmatic patients. The inflammatory nature of asthma helped to establish current diagnosis, management, and potential prevention of the disease. The immunohistopathologic features of asthma include the release of proinflammatory mediators by macrophages and other airway residential cells after stimulation. The subsequent recruitment and accumulation of other inflammatory cells such as neutrophils, eosinophils, TH2-like lymphocytes resulted in further airway inflammation. Airway inflammation contributes to airway hyperresponsiveness, acute bronchoconstriction, edema, mucus plug formation, airflow limitation, and respiratory symptoms. Severe inflammation may cause denudation of airway epithelium and collagen deposition beneath basement membrane. Inadequate airway remodeling can cause persistent airflow limitation. |
本系統中英文摘要資訊取自各篇刊載內容。