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頁籤選單縮合
題 名 | Relationship of Mucopolysaccharide Change, Helicobacter Pylori Infection and Expression of P-53 in Gastric Cancer=黏液變化,幽門桿菌感染及P-53蛋白與胃癌之關係 |
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作 者 | 孫家棟; 吳明賢; 李威傑; 林肇堂; 莊壽洺; | 書刊名 | 慈濟醫學 |
卷 期 | 9:1 1997.03[民86.03] |
頁 次 | 頁27-33 |
分類號 | 415.527 |
關鍵詞 | 胃癌; 黏液; 幽門桿菌; P-53抑癌蛋白; Gastric cancer; Mucopolyssacharide; Helicobacter pylori; P-53 oncoprotein; |
語 文 | 英文(English) |
英文摘要 | 胃癌在癌症死亡率仍佔有一重要地位,所以如何找到一個早期預防之指標和好的 預後因子是迫切需要的。 我們收集民國 81 年至民國 83 年間 112 個接受過胃切除之標本 ,利用勞倫氏分類法將其分成腸形 (63 例 ) 及瀰散形 (49 例 )。 試著由這些標本找出黏 液變化,幽門桿菌,及抑癌蛋白 P-53 之表現與胃癌之關係。我們利用高鐵銨染色法偵測腸 化生情形 (尤其是第三型 ) 免疫染色法測試 P-53 抑癌蛋白之表現。 結果顯示 P-53 陽性 染色可見於 54.5%(61/112) 之標本內,但與年齡, 性別,幽門桿菌或預後沒有統計之關連 ,但明顯增加表現頻率於晚期 (60.7%),腸型 (69.8%) 及賁門 (76.5%) 胃癌。至於第三型 化生合併有胃癌亦明顯有增加 (42.9%, 48/112), 尤其腸形 (66.7%, 42/63) 與瀰散形 (12.2%,6/49) 於統計上更有意義 (p value <0.01)。 初步我們得到的結果是第三型腸形化 生是胃癌 (尤其腸形 ) 之一危險因子,在流行病學上可當成一篩檢因子;而抑癌蛋白 P-53 之表現道出腸形與瀰散形胃癌有兩種不同之致癌機轉,但是否可當成預後因子仍需更多例之 研究;至於幽門桿菌所扮演之角色,本實驗中仍不明確,仍須更進一步努力。 (76.5%) cancer. The coexistence of GC and type III intestinal metaplasia is increased in frequency (42.9%), especially the intestinal type (66.7%, 42/63 vs 12.2%, 6/49; X �� p value < 0.01). Thus we conclude that appearance of type III intestinal metaplasia may represent as a risk factor in intestinal type cancer just as atrophic gastritis to the GC. Overexpression of P-53 protein seems to indicate a different pathway in carcinogenesis while the exact pathway still needs to be elucidated. As for the role of H. pylori, examinations on more specimens are needed to determine. |
本系統中英文摘要資訊取自各篇刊載內容。