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題 名 | 水稻白葉枯病菌培養濾液對於水稻粒線體的影響=Effects of the Culture Filtrate of Xanthomonas Oryzae Pv. Oryzae on Rice Mitochondria |
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作 者 | 李紅玉; 王金生; | 書刊名 | 植物病理學會刊 |
卷 期 | 6:1 1997.03[民86.03] |
頁 次 | 頁25-30 |
分類號 | 434.111 |
關鍵詞 | 活性氧; 跨膜電位; 水稻白葉枯病菌; Active oxygen; Transmembrane potential; Xanthomonas oryzae pv. oryzae; |
語 文 | 中文(Chinese) |
中文摘要 | 李紅玉、王金生. 1996. 水稻白葉枯病菌培養濾液對於水稻粒線體的影響. 植病 會刊 6:25-30. 水稻白葉枯病菌濾液可明顯調節水稻 IR26 粒線體膜的生物物理特性。以親 和程度不同的白葉枯病菌小種 JXO I、 JXO III、 JXO V、 及 JXO III 的毒性基因突變體 M3105,JXO I 的毒性基因突變體 M1103 分別離體處理水稻 IR26 粒線體製備物,能不同程 度地提高粒線體跨膜電位 (△ψ )。濾液濃度實驗結果顯示,水稻白葉枯病菌培養濾液提高 水稻 IR26 粒線體△ψ的能力與其致病能力 (親和程度 ) 呈反相關。 同時,由於粒線體△ ψ的升高,誘發水稻 IR26 細胞質中的 Ca �ワ僕刓u體輸送,導致粒線體內 Ca �Ч@度發生 不同程度的升高。 另外,上述處理還可增加水稻 IR26 粒線體中活性氧 (H �� O �砥BO �� ) 的釋放,且這種增加具有特異性,不親和程度越高的菌株誘導水稻粒線體釋放活性氧的能 力越高。這些實驗結果顯示,水稻 IR26 粒線體膜可能也與細胞膜一樣,帶有能與白葉枯菌 交互作用的識別機制, 通過這種機制可以調節粒線體△ψ、Ca �Ч@度、活性氧釋放等具有 信號功能的變化,進而調節粒線體的物質和能量代謝,並影響水稻防衛基因之表現。 |
英文摘要 | Li, H. and Wang. J. 1997 Effects of the culture filtrate of Xanthomonas oryzae pv. oryzae on rice mitochondria. Plant Pathol. Bull. 6:25-30. (Nanjing Agricultural University, Nanjing, China 210095). The effects of the culture filtrate of Xanthomonas oryzae pv. oryzae on mitochondria of rice cultivar IR26 were investigated. When the rice mitochondria were treated with the culture filtrate of 5 strains of X. o. pv. oryzae, which differed in their pathogenicity on rice, the transmembrane potential (△ψ ) of mitochondria increased rapidly to different levels. Increase in mitochondrial △ψ was negatively correlated with the pathogenicity of X. o. pv. oryzae. Besides, in response to the increase in mitochondrial △ψ, Ca �� moved from the rice cytoplasm to mitochondria and resulted in an increase in [Ca �� ] inside mitochondria. It was also found that the same treatments induced the release of active oxygen (H �� O ��, O �� ) from the mitochondria in a specific way. These results suggest that there may exist a mechanism which underlies the specific interaction between the mitochondrial membrane of rice and X. o. pv. oryzae. Moreover, it may effect a change in the transmembrane potential and induces the release of active oxygen from mitochondria, both of which function in the signal transduction pathway and thereby affect the metabolisms in mitochondria as well as expression of the defense genes. |
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